Definition:
Heart block is a condition in which the conductive tissue of the heart fails to
conduct impulse normally that results in altered rhythm of the heart. A heart block is
a disease in the electrical system of the heart. This is opposed to coronary artery
disease, which is disease of the blood vessels
of the heart. While coronary artery disease can cause angina (chest pain) or myocardial
infarction (heart attack), heart block can cause
lightheadedness, syncope (fainting), and palpitations.
Classifications:
1.
SA block.
2.
AV block.
·
First-degree heart block.
·
Second-degree heart block.
·
Third-degree heart block.
3.
Bundle branch block (Hemi block):
·
Right bundle branch block (RBBB)
·
Left bundle branch block (LBBB).
4.
Purkinjee block.
Aetiology:
[A]Acquired:
·
Focal fibrosis.
·
Rheumatic carditis.
·
Coronary artery disease.
·
Syphilitic carditis.
·
Digitalis toxicity.
·
Atrial fibrillation.
[B]Congenital: VSD.
Clinical
features:
First
degree heart block: Every impulse from atria is conducted to
ventricle with delay.
·
No symptoms.
·
No sign.
·
Only diagnosed by ECG.
Causes:
·
Rheumatic carditis.
·
Diptheric carditis.
·
Digitalis toxicity.
·
Other causes of myocarditis.
·
After MI (esp infection MI)
ECG shows: Prolonged PR
interval (>0.2 sec)
Second
degree heart block: In this condition dropped beats occure
because some impulses from the atrial fail to get through the ventricules.
·
Mobitz type-I: Here progressive
prolongation of P-R interval occurs until a drop beat.
·
Mobitz type-II: Here P-R interval is constant,
but some P waves are not conducted. Pulse is slow and regular.
Third
degree heart block: (Complete heart block) When A-V conduction
fails completely, the atrial & ventricles beat independently (Av
dissociation). Ventricular activity is maintained by an escape rhythm arising
in the bundle of His (narrow QRS complexes) or the distal conducting tissue
(Broad QRS complexes)
Etiology
of complete heart block:
1.
Congenital.
2.
Acquired.
·
Idiopathic fibrosis.
·
MI/ischemia.
·
Inflammation:
Acute (e.g. aortic root abscess in
infective endocarditic)
Chronic (e.g. Sarcoidosis)
·
Trauma (e.g. Cardiac surgery)
·
Drugs (e.g. Digoxin,β-blocker)
Clinical
features:
Symptoms:
1.
May be asymptomatic.
2.
Chest pain.
3.
Breathlessness.
4.
Extreme fatigability.
5.
Stroke Adam’s syndrome.
Signs:
1.
Pulse: Bradycardia (<40/min) which
does not ↑ on exercise.
2.
BP: Systolic=high, Diastolic=Low, wide
pulse pressure.
3.
JVP: Rasied and occasionally showing
cannon wave.
4.
Heat: Varying intensity of the first
heart sound.
5.
ECG findings:
·
P and QRS complexes are independent.
·
P-P interval regular with atrial rate
60-50%.
·
R-R interval regular with ventricular
rate 25-50/min.
·
P-R interval irregular.
·
QRS complex: narrow in AV & wide in
purkinje block.
Management:
[A]AV blocks
complicating acute MI:
a)
If the patient remains well no treatment
is required.
b)
In first degree heart block: no
treatment but the patient must be close monitored.
c)
In 2nd degree heart block:
Atropine (0.6 mg i/v repeated as necessary). If this fails: Temporary
pacemaker.
d)
In 3rd degree (complete)
heart block:
·
Bed rest and oxygen inhalation.
·
Atropine (0.6 mg i/v, repeated as
necessary).
·
Inj. Isoprenaline (1-5 mg) in 500 ml of
5% DA i/v.
·
To prevent recurrence: Use long acting
isoprenaline (30mg) 4 times daily or
·
Correction of acidosis by 7.5% NaHC
.

e)
Complete heart block after infraction:
·
Atropine (0.6 mg) i/v to increase the
heart rate.
·
Temporary pacemaker, or
·
Corticosteroid in bedside for 2 weeks.
·
Glucose & insulin to improve cardiac
function.
f)
Chronic AV block:
·
No treatment: for asymptomatic first
degree or Mobitz type-I 2nd degree AV block.
·
Permanent pacemaker: for asymptomatic
Mobitz type-II 2nd degree or complete heart block.
Reference:
1. Davidson’s Principle and Practice
of Medicine, 21st edition.
2. Wikipedia the free encyclopedia.
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