nts having normal heart rate of 110–150 bpm to the
elderly, who have slower normals.
Signs
and symptoms:
Tachycardia is often asymptomatic. If the heart rate is too high, cardiac
output may fall due to the markedly reduced ventricular filling time.Rapid
rates, though they may be compensating for ischemia elsewhere, increase
myocardial oxygen demand and reduce coronary blood flow, thus precipitating an
ischemic heart or valvular disease Sinus tachycardia accompanying a myocardial
infarction may be indicative of cardiogenic shock.
Cause: Sinus tachycardia is usually a response
to normal physiological situations, such as exercise and an increased
sympathetic tone with increased catecholamine release—stress, fright, flight,
anger. Other causes include:
• Pain
• Hypovolemia wi
• Fever
• Anxiety
• Dehydration
• Malignant hyperthermia
• th hypotension and shock
• Anemia
• Heart failure
• Hyperthyroidism
• Mercury poisoning
• Kawasaki disease
• Pheochromocytoma
• Sepsis
• Pulmonary embolism
• Acute coronary ischemia and myocardial infarction
• Chronic pulmonary disease
• Hypoxia
• Intake of stimulants such as caffeine, nicotine,
cocaine, or amphetamines
• Hyperdynamic circulation
• Electric shock
• Drug withdrawal
Diagnosis: A 12 lead ECG showing sinus tachycardia
Usually apparent on the EKG, but if heart rate is
above 140 bpm the P wave may be difficult to distinguish from the previous T
wave and one may confuse it with a paroxysmal supraventricular tachycardia or
atrial flutter with a 2:1 block. Ways to distinguish the three are:
• Vagal maneuvers (such as carotid sinus massage or
Valsalva's maneuver) to slow the rate and identification of P waves
• administer AV blockers (e.g., adenosine,
verapamil) to identify atrial flutter with 2:1 block
ECG
characteristics
• Rate: Greater than or equal to 100.
• Rhythm: Regular.
• P waves: Upright, consistent, and normal in morphology
(if no atrial disease)
• P–R interval: Between 0.12–0.20 seconds and
shortens with increasing heart rate
• QRS complex: Less than 0.12 seconds, consistent,
and normal in morphology.
Inappropriate
sinus tachycardia
Also known as chronic nonparoxysmal sinus
tachycardia, patients have elevated resting heart rate and/or exaggerated heart
rate in response to exercise. These patients have no apparent heart disease or
other causes of sinus tachycardia. IST is thought to be due to abnormal
autonomic control.
Postural orthostatic tachycardia syndrome Usually
in women with no heart problems, this syndrome is characterized by normal
resting heart rate but exaggerated postural sinus tachycardia with or without
orthostatic hypotension.
Treatment: Not required for physiologic sinus
tachycardia. Underlying causes are treated if present. Acute myocardial
infarction. Sinus tachycardia can present in more than a third of the patients
with AMI but this usually decreases over time. Patients with sustained sinus
tachycardia reflect larger infarcts that are more anterior with prominent left
ventricular dysfunction, associated with high mortality and morbidity.
Tachycardia in the presence of AMI can reduce coronary blood flow and increase
myocardial oxygen demand, aggravating the situation. Beta blockers can be used
to slow the rate, but most patients are usually already treated with beta
blockers as a routine regimen for AMI.
Practically, many studies showed that there is no
need for any treatment.
IST and POTS. Beta blockers are useful if the cause
is sympathetic over activity. If the cause is due to decreased vagal activity,
it is usually hard to treat and one may consider radiofrequency catheter
ablation.
Reference:
1. Davidson’s Principle and Practice of Medicine,
21st edition.
2. Wikipedia the free encyclopedia.
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