Causes
of spontaneous pneumothorax:
1.
Primary:
·
Rupture of subpleural emphysematous
bullae.
·
Rupture of pleural bleb.
·
Rupture of pulmonary end of pleural
adhesion.
2.
Secondary:
·
Rupture of subpleural tubercular focus.
·
Staphylococcal lung abscess.
·
Bronchogenic carcinoma.
·
COPD, asthma, pulmonary infraction.
·
All forms of fibrotic and cystic lung
disease.
Types
of spontaneous pneumothorax:
1.
Closed type.
2.
Open type.
3.
Tension (valvular) type
Pathology:
[A]
Closed type:
The communication
between pleural and lungs seals off as the lung deflates and does not reopen.
The air is gradually absorbed and lung re-expands.
[B] Open type: All abnormal communication is
made between bronchus and pleural space (broncho-pulmonary fistula) the air
pressure in pleural space is equal to atmospheric pressure both on inspiration
and expiration, so it does not responds to water seal drainage and the lung
cannot re-expand.
[C] Tension
(valvular) type:
Most dangerous form.
Communication between pleural and lung persists but is small and acts as one
way valve like action-entry but not escape. Thus pressure within pleural space
rapidly increases causes catastrophic collapse of the lung and mediastinal
displacement.
Clinical
features:
Symptoms:
1.
Sudden sever chest pain usually on the
affected side due to stretching of parietal pleural, radites to the tip of
shoulder and arm.
2.
Increasing dyspnoea, cyanosis in severe
cases:
a) Closed
type: Dysponea is not sever, improves gradually.
b) Open
type: Dysponea is not severe but does not improve.
c) Tension
type: Rapidly progressive severe
dysponoea and accompanied by central cyanosis.
3.
Symptoms of underlying cause.
Signs:
1.
Respiratory rate: Marked increased.
2.
Tachycardia.
3.
There may be hypertension.
4.
Patient may be in shock.
O/E
of respiratory system:
Inspection:
·
Patient is dyspnoeic.
·
Movement of chest is restricted to the
affected side.
·
Intercostal spaces are full.
Palpation:
·
Trachea is shifted to the opposite side.
·
Apex beat may be shifted to opposite
side.
·
Movement of chest diminished on the
affected side.
·
Expansion of chest is reduced (on
affected side)
·
Vocal fremitus is reduced/absent.
Percussion: Hyper-resonance over the affected
area.
Auscultation:
·
Breath sound is vesicular & markedly
diminished.
·
Vocal resonance is reduced.
·
No added sound.
Investigation:
1.
X-ray chest P/A view in erect posture
(most important)
·
Sharply defined collapsed lung margin.
·
Hypertranslucency without any lung
markings in between collapsed lung margin and chest wall.
·
Mediastinal shifting towards opposite
side.
2.
Lung function test.
3.
Sputum for AFB stain & Gram stain.
4.
M.T test.
5.
Complete-blood count: TC, DC, ESR, Hb%.
Management:
[A]
Conservative treatment:
1. Strict
bed rest in propped-up position.
2. Diet-
easily digestible.
3.
inhalation (3-4 litre/min continuous)
![](file:///C:/Users/MYPC~1/AppData/Local/Temp/msohtmlclip1/01/clip_image002.gif)
4. Ensure
easy bowel clearance/movement by laxatives.
5. Broad
spectrum antibiotics.
Then
keep the patient under close observation-if the conservative treatment fails
then we shall go for- Water seal drainage.
[B]
Treatment of underlying cause: depends on type.
1. Closed
type:
·
Small pneumothorax: No active measure,
radiographic observation 2 weekly until re-expansion of the lung is complete.
·
Large pneumothorax: (with
breathlessness): Water seal drainage (intercostals tube drainage) Radiological
observation. Specific chemotherapy if aetiology is TB.
2. Open
type:
Surgical
measure because such cases are amenable to medical treatment, surgery is the
treatment of choice whether it is tuberculous or non-tuberculous.
3. Tension
(valvular) type:
·
Water seal drainage.
·
Radiological observation.
4. Recurrent
spontaneous pneumothorax: Obliteration of at least one pleural
space (Pleurodesis) by introducing irritant substance such as tetracycline,
Kaolin, bleomycin or by pleurectomy at thoractomy.
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